Hypoglycemic Effect of Foeniculum vulgare Mill. Fruit on Dexamethasone Induced Insulin Resistance Rats
VR Dongare1*, AU Arvindekar2 and CS Magadum3
1Appasaheb Birnale College of Pharmacy, Sangli
2Department
of Biochemistry, Shivaji
University, Kolhapur.
3Ashokrao Mane College of Pharmacy, Pethvadgaon. (M.S) India
ABSTRACT:
Aim:
- To assess the hypoglycemic effect of Foeniculum vulgare Mill. Fruit on dexamethasone
induced insulin resistance rats.
Method: - Wistar male rats (n=5) were made insulin resistance by
administering dexamethasone injection subcutaneously
to the overnight fasted rats and continue till the end of the experiment along
with methanolic extract of Foeniculum vulgare Mill. Fruit for test and pioglitazone for std. At the end of the experimental period
i.e. on day 11th the animals anesthetize with ether, blood was
collected by retroobital puncture and serum separated
for the estimation of glucose, triglycerides, cholesterol, insulin. The animals
were weighed at the beginning and at the end of experimental period. The liver
and muscle glycogen was also measured.
Results: - Methanolic extract of Foeniculum vulgare Mill. Fruit lowers blood
glucose, triglycerides, cholesterol and increase liver and muscle glycogen in dexamethasone induced insulin resistance rats.
Conclusion:
The present study was indicating that Foeniculum vulgare Mill.
Fruit showed significant glucose and triglyceride lowering activities as
compared with diabetic control. In comparison with standard pioglitazone,
Foeniculum vulgare Mill.
Fruit showed comparable serum glucose lowering and better triglyceride lowering
activity.
KEYWORDS: Insulin
resistance, Foeniculum vulgare, Dexamethasone, Blood glucose,
INTRODUCTION:
Diabetes
mellitus is disorder of carbohydrate, fat, protein metabolism attributed to
diminished production of insulin or insulin resistance.1
Diabetes is
a global disease with a huge adverse impact on health and mortality,
particularly from cardiovascular disorders. It occurs at any time of life from
infancy to old age. Type 2 diabetes is primarily a life style disorder, which
accounts for around 90% of diabetes cases and increasing at an astonishing
rate, particularly in developing countries like India. In 1995, it has been
estimated that around 135 million people had this condition, and this may
increase to as many as 300 million by the year 2025.2
It is
characterized by abnormalities in carbohydrate, lipid and lipoprotein
metabolism which not only lead to hyperglycemia but also cause many
complications, such as hyperlipidemia, hyperinsulinemia, hypertension and atherosclerosis.3
Coronary, cerebrovascular and peripheral
vascular diseases are the principal causes of morbidity and the mortality in
type 2 diabetes mellitus. The accelerated macro vascular disease in type2
diabetes mellitus is due to partly in increased incidence of cardiovascular
risk factor such as hypertension, obesity and dyslipidemia.
Type 2 diabetes mellitus is associated with an increased incidence of micro and
macroangiopathy and hypertension.
These disorders may,
together with other risk factors, such as hyperlipidemia,
oxidant stress and insulin resistance state alters endothelial function and
consequently triggers the start of atherosclerosis in type 2 diabetes mellitus.
Endothelial function
is more altered in type 2 diabetic patient than in type1 diabetic patient, even
though the blood glucose control may not be significantly different. A longer
period of undetected blood abnormalities, lipid alterations or decreased
insulin sensitivity could be involved. Biochemical changes are activated by high
blood glucose concentration and can explain the adverse effects of hypoglycemia
on the retina, lens, nerves and also on endothelium.4
Increasing evidence
from both experimental and clinical studies suggests that oxidative stress
plays a major role in the pathogenesis of diabetes mellitus. Free radicals are
formed disproportionately in diabetes by glucose oxidation, non enzymatic glycation of proteins and the subsequent oxidative
degradation of glycated proteins. Recent experimental
and clinical studies have uncovered new insights in to the role of oxidative
stress in diabetic complications, suggesting a different and innovative
approach to a possible “causal” antioxidant therapy e.g. flavonoids.5
MATERIALS
AND METHODS:-
1]
Preparation of extract-
The
air dried powdered Foeniculum vulgare Mill. Fruit extracted in soxhlet extraction assemblyby
using methanol.6
2]
Acute oral toxicity study-
The acute oral
toxicity study was studied as per the OECD guideline 425 in Wistar
rats maintained under standard conditions. Acute toxicity for methanolic extract of Foeniculum vulgare Mill. Fruit was carried
out using groups of three wistar rats by
administering a dose 2000 mg/kg, in normal saline orally while the control
received only the vehicle.7
3]
Antidiabetic activity:-
Animals:-
Wistar rats
weighed between150-250 which was bred in the Department of pharmacology
A.B.C.P. Sangli. The animals were housed under well
ventilated natural conditions in large cage. The animals were made insulin
resistance by administering dexamethasone
subcutaneously and hypoglycemic activity of methanolic
extract of Foeniculum vulgare Mill. Fruit was assessed.
Dexamethasone induced insulin resistance model-
Animals were divided
in to following manner- for Dexamethasone induced insulin resistance model-
1] Normal control |
Oral
saline |
2] Dexamethasone control |
Dexamethasone sodium
phosphate 10mg/kg once daily, s.c. |
3] Test |
Dexamethasone sodium
phosphate + methanolic extract (200mg/kg b.w., p.o.) |
4] Std. |
Dexamethasone sodium
phosphate + Pioglitazone
(30mg/kg, p.o.) |
Method: Dexamethasone induced insulin resistance model-
In groups 2-4, dexamethasone was injected subcutaneously to the overnight
fasted rats and continues till the end of the experiment along with methanolic extract for test and pioglitazone
for std. At the experimental period i.e. on day 11th the animals
anesthetize with ether, blood was collected by retroorbital
puncture and serum separated for the estimation of glucose, triglyceride,
cholesterol, insulin. The animals were weighed at the beginning and at the end
of the experimental period. The liver and muscle glycogen was also measured.
Serum insulin was estimated by ELISA method using ELISA kit.8
Statistical
Analysis:
All the results were
expressed as mean SEM and the data were analyzed using one–way ANOVA followed
by Dunnett’s multiple comparison post hock test
P-values <0.01were considered significant9
RESULTS:
Acute
toxicity study:-
The groups were
observed mortality and behavioral changes during 48hrs. The LD50 of methanolic extract
of Foeniculum vulgare in rats
by oral route was found to be 200mg/Kg.
Antidiabetic activity:-
Biochemical
parameters |
Normal Control |
Diabetic
Control |
Test |
Std |
Blood
glucose |
90.2± 2.57** |
202.2± 3.39* |
98± 1.4 |
100.2 ±1.28 |
Cholesterol |
72.3± 0.64** |
108.4± 1.03* |
88.7 ±0.46 |
85.76 ±0.45 |
Triglycerides |
100.9± 2.33** |
201.72 ±2.55* |
149.12±1.3 |
133.5 ±0.51 |
HDL
Cholesterol |
38.7± 0.60** |
21.79± 0.27* |
35.27 ±0.28 |
33.47 ±0.21 |
LDL |
13.42± 0.68** |
50.46± 2.08* |
26.60 ±0.28 |
25.75 ±0.74 |
VLDL |
20.18± 0.46** |
40.34± 0.51* |
29.82 ±0.27 |
26.71 ±0.10 |
Insulin |
25.67± 0.36** |
42.51± 0.70* |
28.78 ±0.54 |
27.92 ±0.39 |
Liver
glycogen |
35.64± 0.77** |
22.284 ±1.35* |
32.9± 1.47 |
36.5 ±0.50 |
Muscle
glycogen |
6.12± 0.23** |
3.296± 0.34* |
5.8± 0.43 |
6.2 ±0.24 |
Table showing Foeniculum vulgare
significantly lowers blood glucoseand triglycerides.
Values are mean ±SEM; n=5 in each group
*p<0.01 when compared with control. **p<0.01 when compared with normal.
DISCUSSION:-
The results of this
study demonstrated that dexamethasone increases
triglyceride levels, causing an imbalance in lipid metabolism leading to hyperlipidemia and increase in glucose levels leading to
hyperglycemia.10
The use of medicinal
plants has a long folk for the treatment of diabetes mellitus, (11) currently
available synthetic antidiabetic agent produce
serious side effects like hypoglycemic coma. Thus it is possible to develop
newer drugs, lesser side effects, and better efficiency at lowered cost.
In summary, the
present study demonstrated that type II diabetes mellitus leads to insulin
resistance and elevated level of cholesterol, triglycerides and LDL, which can
be significantly managed by methanolic extract of Foeniculum vulgare mill.
Fruit.
ACKNOWLEDGEMENT:
The author
is thankful to Principal shri D. D. Chougule, Appasaheb Birnale College of Pharmacy , Sangli
for providing facilities.
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Received on
07.07.2009
Accepted on
10.09.2009
© A&V
Publication all right reserved
Research
Journal of Pharmacognosy and
Phytochemistry. 2(2): March -April 2010, 163-165